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Monday, October 19, 2009

"Depression" (recent Ggl sch)

Oka last page checked was 13

[BOOK] Manic-depressive illness: bipolar disorders and recurrent depression


FK Goodwin, KR Jamison - 2007 - books.google.com
Page 1. SECOND EDITION MANIC- DEPRESSIVE ILLNESS Bipolar Disorders and Recurrent
Depression FREDERICK K. GOODWIN d- KAY REDFIELD JAMISON Page 2. ...
Cited by 102 - Related articles - All 6 versions


Abstract: http://content.nejm.org/cgi/content/abstract/354/11/1130
Maintenance Treatment of Major Depression in Old Age

6 pages of abstracts on major depression and suicide (the above)

Culture and depression - Arthur Klein, MD In many parts of Chinese society, the experience of depression is physical rather than psychological. Many depressed Chinese people do not report feeling sad, but rather express boredom, discomfort, feelings of inner pressure, and symptoms of pain, dizziness, and fatigue. These culturally coded symptoms may confound diagnosis among Chinese immigrants in the United States, many of whom find the diagnosis of depression morally unacceptable and experientially meaningless; this cultural pattern changes over time but continues to diverge significantly from the experiences of other groups. The pattern of somatization may be unfamiliar to U.S. clinicians and may further complicate the concept


Sleep and Depression.
Tsuno, Norifumi; Besset, Alain; Ritchie, Karen
Journal of Clinical Psychiatry. Vol 66(10), Oct 2005, 1254-1269.
Background: Of all the psychiatric disorders associated with insomnia, depression is the most common. It has been estimated that 90% of patients with depression complain about sleep quality. Since the first reports of short rapid eye movement (REM) latency in depressed patients and of the effect of sleep deprivation on depression in the 1970s, numerous sleep studies have provided extensive observations and theoretical hypotheses concerning the etiology and pathophysiology of depression. The aim of this review is to summarize knowledge regarding the relationships between sleep and depression. Data Sources and Selection: MEDLINE and PsycINFO searches of the literature published in English or French between 1964 and 2005 that examined the relationships between sleep disturbance and depression were conducted. Search terms used were depression, depressive disorder, affective disorder, mood disorders, seasonal affective disorder, sleep, sleep disorders, insomnia, REM, polysomnography, sleep deprivation, electroencephalography, PET, SPECT, and fMRI. Data Synthesis: Two hundred five papers were identified and selected and then integrated into the following categories: sleep architecture, antidepressive therapies, age- and gender-associated differences, functional imaging results, and sleep-related hypotheses explaining the pathophysiology of depression. Conclusion: Numerous studies provide findings indicating the remarkable relationship between sleep alterations and depression. Although the existing hypotheses are not likely to explain all aspects of the sleep alterations in depression, each may be worth being maintained for refinements of pathophysiologic models of depression as new data accumulate. Further research taking into account the heterogeneity of depressive disorder and linking the different areas of research is needed to develop more comprehensive theoretical models and new therapies for depression. (PsycINFO Database Record (c) 2009 APA, all rights reserved)http://psycnet.apa.org/?fa=main.doiLanding&uid=2006-10343-008
Role of neurotrophic factors in depression
Purchase the full-text article

Eero Castréna, E-mail The Corresponding Author, Vootele Võikara and Tomi Rantamäkia

aSigrid Jusélius Laboratory of Molecular Neuroscience, Neuroscience Center, University of Helsinki, PO Box 56, 00014 Helsinki, Finland


Available online 17 October 2006.

Major depression is associated with reduced volumes in the hippocampus and prefrontal cortex, whereas antidepressant treatments promote several forms of neuronal plasticity, including neurogenesis, synaptogenesis and neuronal maturation, in the hippocampus. Several neurotrophic factors are associated with depression or antidepressant action. Stress suppresses brain-derived neurotrophic factor (BDNF) synthesis in the hippocampus, at least partially through a sustained modification of chromatin structure. Essentially all antidepressant treatments increase BDNF synthesis and signaling in the hippocampus and prefrontal cortex. This signaling is required for the behavioral effects of antidepressant drugs in rodents, and increased BDNF levels in the hippocampus mimic the behavioral effects of antidepressants. However, injection of BDNF into the mesolimbic dopamine pathway produces an opposing depression-like response. One hypothesis emerging from these data proposes that mood disorders reflect failed function of critical neuronal networks, whereas a gradual network recovery through activity-dependent neuronal plasticity induces the antidepressant effect. Neurotrophic factors themselves do not control mood, but they act as necessary tools in the activity-dependent modulation of networks, the physiological function of which determines how a plastic change influences mood.

http://www.sciencedirect.com/science?_ob=ArticleURL&_udi=B6W7F-4M4KPYX-1&_user=10&_rdoc=1&_fmt=&_orig=search&_sort=d&_docanchor=&view=c&_acct=C000050221&_version=1&_urlVersion=0&_userid=10&md5=d041538ba9a1baf6ede34fbac26ce8f5


NFLUENTIAL PUBLICATION

Assessing and Managing Depression in the Terminally Ill Patient

Susan D. Block, M.D. for the American College of Physicians–American Society of Internal Medicine End-of-Life Care Consensus Panel

Psychological distress often causes suffering in terminally ill patients and their families and poses challenges in diagnosis and treatment. Increased attention to diagnosis and treatment of depression can improve the coping mechanisms of patients and families. This paper reviews the clinical characteristics of normal grief and clinical depression and explains strategies for differential diagnosis. Although some literature discusses the psychological issues facing elderly patients and terminally ill patients with cancer, less is known about patients with end-stage pulmonary, cardiac, renal, and neurologic disease. Data on the effectiveness of interventions in terminally ill patients are lacking. Treatment recommendations in this paper represent extrapolations from existing literature and expert opinion. Diagnosing and treating depression in terminally ill patients involve unique challenges. Evidence of hopelessness, helplessness, worthlessness, guilt, and suicidal ideation are better indicators of depression in this context than neurovegetative symptoms. Although terminally ill patients often have suicidalthoughts, they are usually fleeting. Sustained suicidal ideation should prompt a comprehensive evaluation. Clinicians should have a low threshold for treating depression in terminally ill patients. Psychostimulants, because of their rapid onset of action, are useful agents and are generally well tolerated. Selective serotonin reuptake inhibitors and tricyclic antidepressants may also be used. Psychological interventions—including eliciting concerns and conveying the potential for connection, meaning, reconciliation, and closure in the dying process—can also facilitate coping.

(Reprinted with permission from the Annals of Internal Medicine 2000;132:209–218; © 2000 American College of Physicians–American Society of Internal Medicine)

http://focus.psychiatryonline.org/cgi/content/abstract/3/2/310

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Cognitive Therapy vs Medications in the Treatment of Moderate to Severe Depression

Robert J. DeRubeis, PhD; Steven D. Hollon, PhD; Jay D. Amsterdam, MD; Richard C. Shelton, MD; Paula R. Young, PhD; Ronald M. Salomon, MD;John P. O’Reardon, MD; Margaret L. Lovett, MEd; Madeline M. Gladis, PhD; Laurel L. Brown, PhD; Robert Gallop, PhD

Arch Gen Psychiatry. 2005;62:409-416.

WHOLE ARTICLE GLORY BE!!!!

here's the source: http://archpsyc.ama-assn.org/cgi/content/full/62/4/409

SEE P. 11 this is where I left off

Delivering interventions for depression by using the internet: randomised controlled trial

- bmj.com
H Christensen, KM Griffiths, AF Jorm - British Medical Journal, 2004 - bmj.com
... Primary care. Delivering interventions for depression by using the internet: randomised
controlled trial. ... with depression do not receive adequate treatment. ...
Cited by 240 - Related articles - BL Direct - All 12 versions

Effects of citalopram and interpersonal psychotherapy on depression in patients with …


F Lesperance, N Frasure-Smith, D Koszycki, … - Jama, 2007 - Am Med Assoc
... Effects of Citalopram and Interpersonal Psychotherapy on Depression in
Patients With Coronary Artery Disease The Canadian Cardiac ...
Cited by 87 - Related articles - Get at CISTI - All 7 versions

Efficacy of quetiapine monotherapy in bipolar I and II depression: a double-blind, placebo- …


ME Thase, W Macfadden, RH Weisler, W … - Journal of clinical psychopharmacology, 2006 - journals.lww.com
... Original Contributions. Efficacy of Quetiapine Monotherapy in Bipolar I and II
Depression: A Double-blind, Placebo-controlled Study (The BOLDER II Study). ...
Cited by 111 - Related articles - Get at CISTI - All 5 versions

Prediction from low birth weight to female adolescent depression: a test of competing …

- angrylapdog.com
EJ Costello, C Worthman, A Erkanli, A Angold - Archives of general psychiatry, 2007 - Am Med Assoc
... Prediction From Low Birth Weight to Female Adolescent Depression A Test of Competing
Hypotheses. ... Its role as a predictor of depression is unclear. ...
Cited by 51 - Related articles - Get at CISTI - All 9 versions

Efficacy of ECT in depression: a meta-analytic review


D Pagnin, V de Queiroz, S Pini, GB Cassano - Focus, 2008 - Am Psychiatric Assoc
... INFLUENTIAL PUBLICATIONS. Efficacy of ECT in Depression: A Meta-Analytic Review. ...
The definition and meaning of treatment-resistant depression. J Clin Psychiatry. ...
Cited by 62 - Related articles - Get at CISTI - All 5 versions

Modulation of cortical-limbic pathways in major depression: treatment-specific effects of …

- wisc.edu [PDF]
K Goldapple, Z Segal, C Garson, M Lau, P … - Archives of General Psychiatry, 2004 - Am Med Assoc
... Modulation of Cortical-Limbic Pathways in Major Depression ... Clinical response
was monitored weekly using the Beck Depression Inventory. ...
Cited by 341 - Related articles - Get at CISTI - All 11 versions

Life events, first depression onset and the serotonin transporter gene

- rcpsych.org [PDF]
KAY Wilhelm, PB Mitchell, H Niven, A Finch, … - The British Journal of Psychiatry, 2006 - RCP
... interactionin a cohort withlongitudinal dataforlife events, experience of dataforlife
events, experience of depression, parentalbondingand depression ...
Cited by 109 - Related articles - Get at CISTI - All 5 versions

Screening and case-finding instruments for depression: a meta-analysis

- ecmaj.ca
S Gilbody, T Sheldon, A House - Canadian Medical Association Journal, 2008 - ecmaj.ca
... Research. Screening and case-finding instruments for depression: a meta-analysis. ...
Screening and case finding instruments for depression. ...
Cited by 76 - Related articles - Get at CISTI - All 15 versions

… magnetic stimulation in the acute treatment of major depression: a multisite randomized …

- butler.org [PDF]
JP O'Reardon, HB Solvason, PG Janicak, S … - Biological Psychiatry, 2007 - Elsevier
... Biological Psychiatry Volume 62, Issue 11, 1 December 2007, Pages 1208-1216
Depression: New Perspectives on Treatment and Etiology. ...
Cited by 85 - Related articles - Get at CISTI - All 21 versions

The association of depression and anxiety with medical symptom burden in patients with …


W Katon, EHB Lin, K Kroenke - General Hospital Psychiatry, 2007 - Elsevier
... All rights reserved. Symptom Research. The association of depression and anxiety
with medical symptom burden in patients with chronic medical illness


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